Hello, I am looking for someone to write an essay on Inadequate nutrition and cognitive decline. It needs to be at least 1750 words.Download file to see previous pages... This course study will dwell

Hello, I am looking for someone to write an essay on Inadequate nutrition and cognitive decline. It needs to be at least 1750 words.

This course study will dwell on the subject of age-related cognitive decline and nutrition as a contributing factor. The lecture will cover the normal and the non-normal aging process of individuals and in particular how nutrients add up to the neurological well-being of the elderly. The hypothesis in this discussion is that: nutritional imbalance is a resultant factor in cognitive decline. We shall review related studies and literature to support this hypothesis which aims to create awareness among all individuals to continue trying to find the nearest approach to cure and prevention of cognitive abnormalities.As the human body tends to grow with age, a certain amount of memory decline is felt which includes the minute reductions or delay in thought processing. For others though, a magnitude of reduction beyond what may be considered "normal" and are relentlessly progressive, robbing individuals of their memories, intellect, and eventually their abilities to recognize close members of their families may concurrently result to a certain cognitive decline. The increasing risk of demented elderly adults created the idea of preventing or reversing neurological disorders associated with cognitive decline. The results of the clinical investigative studies revealed according to Amador(1998) the discovery of vitamin deficiencies causing neurological and psychiatric symptoms. Further, Amador added that the potentials of Vit.B(cobalamin) deficiency has been overlooked as a treatable cause of reversible dementia. Markle(1996)critically reviewed the alteration in thought processes associated with Vit. B deficiencies and discovered several causes that contribute its loss in the bodies among elderly patients. Quadri et al(2004)further added elevated homocysteine concentration results to folate deficiency that aggravates the early risk factor for cognitive decline in elderly individuals. However Under Wright's(2000) free-radical theory of aging, he provided that the effects of "oxidative damage" are responsible for much aging particularly of the premature kind. Wright also suggested based on animal studies that diets high in anti-oxidant-rich foods may be beneficial in slowing ARCD. Among people aged 65 and older, high vitamin C and beta-carotene levels have been associated with better memory performance. Zagaria(2002)further explained that these nutrients as explained may be markers for other dietary factors responsible for protection against cognitive disorders. In most cases when physical activity is diminished, cellular metabolism is likewise affected which results to mal-absorption of nutrients that are needed by the body. Inadequate dietary intake, intake of drugs that inhibit B12 absorption, increased cobalamin excretion that can occur according to Amador in the presence of normal hematological studies. A detailed description on the role of specific nutrients in age-related cognitive decline will be discussed which includes: Vit.B (cobalamin) and folates. antioxidants and other food products.

Vit.B(cobalamin)

Andel et al (2005) noticed strong evidence of connection between the low levels of vitamin B12 and warned that this is a common nutritional deficiency in the Western world. Commonly present in meat, poultry, fish, eggs and dairy products adults over 50 are mostly lacking than most people due to atropic gastritis whereby B12 absorption in the small intestine is reduced.However, it should be noted that certain acidic elements are persistent in the stomach of the elderly despite age, it is unusually unthinkable to blame the lack of needed nutrients to gastritis.

Teunisse et al(1996)related that cobalamin is an essential vitamin for hematopoiesis and the maintenance of neurologic and psychiatric health. Since it is not synthesized in the body, animals must obtain Vit.B 12 from their diet. Vit.B12 works by aiding in cellular formation and longevity.